Jeremiah Peiffer

Professor; Dean of Graduate Research

Dementia | exercise High-intensity | fitness Blood | flow restriction | cycling exercise | performance

About Me

Professor Jeremiah Peiffer completed his Doctor of Philosophy investigating the impact of post-exercise cold water immersion on exercise performance in 2018 from Edith Cowan University. He has subsequently had roles as a Post-Doctoral Researcher within the Exercise Medicine Research Institute at Edith Cowan University and as a researcher and Department Lead of Exercise Science at Murdoch University. Jeremiah is currently the Dean of Graduate Research at Murdoch University. With a broad research interest, he has published over 100 peer-reviewed scientific papers, secured over $1.0 million in research funding, established the Centre for Healthy Ageing at Murdoch University, and successfully mentored 18 doctoral candidates to complete their degrees. His current research focuses on the delivery of exercise to enable individuals to age healthy by retaining their physical and cognitive faculties. 

Organisational Affiliations

Centre for Healthy Ageing, Health Futures Institute, Murdoch University

Dean of Graduate Studies, Research and Innovation Office, Murdoch University

Education

Kinesiology (Exercise Science)
01/2002–01/2004, Master of Science (MSc), California State University, Sacramento, United States
Kinesiology (Exercise Science)
01/2002–01/2004, Master of Science (MSc), California State University, Sacramento, United States

Systemic hypoxia has a larger effect on reducing the external load at lower exercise intensity during .

by Siu Nam Li, Peter Peeling, Brendan R Scott, Jeremiah J Peiffer, Alex Shaykevich and Olivier Girard

Published 2024

European journal of sport science, Early View

The effects of acute hypoxic exposure on mechanical output and internal responses during cycling with heart rate (HR) clamped at lactate thresholds 1 and 2 (LT1 and LT2, respectively) were investigated. On separate days, 12 trained males cycled for 15 min at a clamped HR corresponding to LT1 and LT2 under normoxic or hypoxic conditions (simulated altitude of ∼3500 m and inspired oxygen fraction of 13.6%). Power output (PO), arterial oxygen saturation, ventilatory and perceptual responses were measured every 3 min, with metabolic response assessed pre- and post-exercise. At LT1, PO was consistently lower in hypoxia compared to normoxia (p < 0.01). At LT2, PO was not different between normoxia and hypoxia at 3 and 6 min (both p > 0.42) but was significantly lower in hypoxia at 9, 12 and 15 min (all p < 0.04). Overall, hypoxia induced a greater decrease in PO at LT1 (-33.3% ± 11.3%) than at LT2 (-18.0 ± 14.7%) compared to normoxia. Ventilatory, perceptual and metabolic responses were influenced by exercise intensity (all p < 0.01) but not environmental conditions (all p > 0.17). A simulated altitude of ∼3500 m is more effective in reducing cycling PO at LT1 than LT2 during HR clamped cycling while maintaining other internal loads. Therefore, normobaric hypoxia provides a greater benefit via a larger decrease in the mechanical constraints of exercise at lower exercise intensities.

Systemic hypoxia has a larger effect on reducing the external load at lower exercise intensity during .

by Siu Nam Li, Peter Peeling, Brendan R Scott, Jeremiah J Peiffer, Alex Shaykevich and Olivier Girard

Published 2024

European journal of sport science, Early View

The effects of acute hypoxic exposure on mechanical output and internal responses during cycling with heart rate (HR) clamped at lactate thresholds 1 and 2 (LT1 and LT2, respectively) were investigated. On separate days, 12 trained males cycled for 15 min at a clamped HR corresponding to LT1 and LT2 under normoxic or hypoxic conditions (simulated altitude of ∼3500 m and inspired oxygen fraction of 13.6%). Power output (PO), arterial oxygen saturation, ventilatory and perceptual responses were measured every 3 min, with metabolic response assessed pre- and post-exercise. At LT1, PO was consistently lower in hypoxia compared to normoxia (p < 0.01). At LT2, PO was not different between normoxia and hypoxia at 3 and 6 min (both p > 0.42) but was significantly lower in hypoxia at 9, 12 and 15 min (all p < 0.04). Overall, hypoxia induced a greater decrease in PO at LT1 (-33.3% ± 11.3%) than at LT2 (-18.0 ± 14.7%) compared to normoxia. Ventilatory, perceptual and metabolic responses were influenced by exercise intensity (all p < 0.01) but not environmental conditions (all p > 0.17). A simulated altitude of ∼3500 m is more effective in reducing cycling PO at LT1 than LT2 during HR clamped cycling while maintaining other internal loads. Therefore, normobaric hypoxia provides a greater benefit via a larger decrease in the mechanical constraints of exercise at lower exercise intensities.

Systemic hypoxia has a larger effect on reducing the external load at lower exercise intensity during .

by Siu Nam Li, Peter Peeling, Brendan R Scott, Jeremiah J Peiffer, Alex Shaykevich and Olivier Girard

Published 2024

European journal of sport science, Early View

The effects of acute hypoxic exposure on mechanical output and internal responses during cycling with heart rate (HR) clamped at lactate thresholds 1 and 2 (LT1 and LT2, respectively) were investigated. On separate days, 12 trained males cycled for 15 min at a clamped HR corresponding to LT1 and LT2 under normoxic or hypoxic conditions (simulated altitude of ∼3500 m and inspired oxygen fraction of 13.6%). Power output (PO), arterial oxygen saturation, ventilatory and perceptual responses were measured every 3 min, with metabolic response assessed pre- and post-exercise. At LT1, PO was consistently lower in hypoxia compared to normoxia (p < 0.01). At LT2, PO was not different between normoxia and hypoxia at 3 and 6 min (both p > 0.42) but was significantly lower in hypoxia at 9, 12 and 15 min (all p < 0.04). Overall, hypoxia induced a greater decrease in PO at LT1 (-33.3% ± 11.3%) than at LT2 (-18.0 ± 14.7%) compared to normoxia. Ventilatory, perceptual and metabolic responses were influenced by exercise intensity (all p < 0.01) but not environmental conditions (all p > 0.17). A simulated altitude of ∼3500 m is more effective in reducing cycling PO at LT1 than LT2 during HR clamped cycling while maintaining other internal loads. Therefore, normobaric hypoxia provides a greater benefit via a larger decrease in the mechanical constraints of exercise at lower exercise intensities.

Systemic hypoxia has a larger effect on reducing the external load at lower exercise intensity during .

by Siu Nam Li, Peter Peeling, Brendan R Scott, Jeremiah J Peiffer, Alex Shaykevich and Olivier Girard

Published 2024

European journal of sport science, Early View

The effects of acute hypoxic exposure on mechanical output and internal responses during cycling with heart rate (HR) clamped at lactate thresholds 1 and 2 (LT1 and LT2, respectively) were investigated. On separate days, 12 trained males cycled for 15 min at a clamped HR corresponding to LT1 and LT2 under normoxic or hypoxic conditions (simulated altitude of ∼3500 m and inspired oxygen fraction of 13.6%). Power output (PO), arterial oxygen saturation, ventilatory and perceptual responses were measured every 3 min, with metabolic response assessed pre- and post-exercise. At LT1, PO was consistently lower in hypoxia compared to normoxia (p < 0.01). At LT2, PO was not different between normoxia and hypoxia at 3 and 6 min (both p > 0.42) but was significantly lower in hypoxia at 9, 12 and 15 min (all p < 0.04). Overall, hypoxia induced a greater decrease in PO at LT1 (-33.3% ± 11.3%) than at LT2 (-18.0 ± 14.7%) compared to normoxia. Ventilatory, perceptual and metabolic responses were influenced by exercise intensity (all p < 0.01) but not environmental conditions (all p > 0.17). A simulated altitude of ∼3500 m is more effective in reducing cycling PO at LT1 than LT2 during HR clamped cycling while maintaining other internal loads. Therefore, normobaric hypoxia provides a greater benefit via a larger decrease in the mechanical constraints of exercise at lower exercise intensities.

Systemic hypoxia has a larger effect on reducing the external load at lower exercise intensity during .

by Siu Nam Li, Peter Peeling, Brendan R Scott, Jeremiah J Peiffer, Alex Shaykevich and Olivier Girard

Published 2024

European journal of sport science, Early View

The effects of acute hypoxic exposure on mechanical output and internal responses during cycling with heart rate (HR) clamped at lactate thresholds 1 and 2 (LT1 and LT2, respectively) were investigated. On separate days, 12 trained males cycled for 15 min at a clamped HR corresponding to LT1 and LT2 under normoxic or hypoxic conditions (simulated altitude of ∼3500 m and inspired oxygen fraction of 13.6%). Power output (PO), arterial oxygen saturation, ventilatory and perceptual responses were measured every 3 min, with metabolic response assessed pre- and post-exercise. At LT1, PO was consistently lower in hypoxia compared to normoxia (p < 0.01). At LT2, PO was not different between normoxia and hypoxia at 3 and 6 min (both p > 0.42) but was significantly lower in hypoxia at 9, 12 and 15 min (all p < 0.04). Overall, hypoxia induced a greater decrease in PO at LT1 (-33.3% ± 11.3%) than at LT2 (-18.0 ± 14.7%) compared to normoxia. Ventilatory, perceptual and metabolic responses were influenced by exercise intensity (all p < 0.01) but not environmental conditions (all p > 0.17). A simulated altitude of ∼3500 m is more effective in reducing cycling PO at LT1 than LT2 during HR clamped cycling while maintaining other internal loads. Therefore, normobaric hypoxia provides a greater benefit via a larger decrease in the mechanical constraints of exercise at lower exercise intensities.

Systemic hypoxia has a larger effect on reducing the external load at lower exercise intensity during .

by Siu Nam Li, Peter Peeling, Brendan R Scott, Jeremiah J Peiffer, Alex Shaykevich and Olivier Girard

Published 2024

European journal of sport science, Early View

The effects of acute hypoxic exposure on mechanical output and internal responses during cycling with heart rate (HR) clamped at lactate thresholds 1 and 2 (LT1 and LT2, respectively) were investigated. On separate days, 12 trained males cycled for 15 min at a clamped HR corresponding to LT1 and LT2 under normoxic or hypoxic conditions (simulated altitude of ∼3500 m and inspired oxygen fraction of 13.6%). Power output (PO), arterial oxygen saturation, ventilatory and perceptual responses were measured every 3 min, with metabolic response assessed pre- and post-exercise. At LT1, PO was consistently lower in hypoxia compared to normoxia (p < 0.01). At LT2, PO was not different between normoxia and hypoxia at 3 and 6 min (both p > 0.42) but was significantly lower in hypoxia at 9, 12 and 15 min (all p < 0.04). Overall, hypoxia induced a greater decrease in PO at LT1 (-33.3% ± 11.3%) than at LT2 (-18.0 ± 14.7%) compared to normoxia. Ventilatory, perceptual and metabolic responses were influenced by exercise intensity (all p < 0.01) but not environmental conditions (all p > 0.17). A simulated altitude of ∼3500 m is more effective in reducing cycling PO at LT1 than LT2 during HR clamped cycling while maintaining other internal loads. Therefore, normobaric hypoxia provides a greater benefit via a larger decrease in the mechanical constraints of exercise at lower exercise intensities.

Systemic hypoxia has a larger effect on reducing the external load at lower exercise intensity during .

by Siu Nam Li, Peter Peeling, Brendan R Scott, Jeremiah J Peiffer, Alex Shaykevich and Olivier Girard

Published 2024

European journal of sport science, Early View

The effects of acute hypoxic exposure on mechanical output and internal responses during cycling with heart rate (HR) clamped at lactate thresholds 1 and 2 (LT1 and LT2, respectively) were investigated. On separate days, 12 trained males cycled for 15 min at a clamped HR corresponding to LT1 and LT2 under normoxic or hypoxic conditions (simulated altitude of ∼3500 m and inspired oxygen fraction of 13.6%). Power output (PO), arterial oxygen saturation, ventilatory and perceptual responses were measured every 3 min, with metabolic response assessed pre- and post-exercise. At LT1, PO was consistently lower in hypoxia compared to normoxia (p < 0.01). At LT2, PO was not different between normoxia and hypoxia at 3 and 6 min (both p > 0.42) but was significantly lower in hypoxia at 9, 12 and 15 min (all p < 0.04). Overall, hypoxia induced a greater decrease in PO at LT1 (-33.3% ± 11.3%) than at LT2 (-18.0 ± 14.7%) compared to normoxia. Ventilatory, perceptual and metabolic responses were influenced by exercise intensity (all p < 0.01) but not environmental conditions (all p > 0.17). A simulated altitude of ∼3500 m is more effective in reducing cycling PO at LT1 than LT2 during HR clamped cycling while maintaining other internal loads. Therefore, normobaric hypoxia provides a greater benefit via a larger decrease in the mechanical constraints of exercise at lower exercise intensities.

Systemic hypoxia has a larger effect on reducing the external load at lower exercise intensity during .

by Siu Nam Li, Peter Peeling, Brendan R Scott, Jeremiah J Peiffer, Alex Shaykevich and Olivier Girard

Published 2024

European journal of sport science, Early View

The effects of acute hypoxic exposure on mechanical output and internal responses during cycling with heart rate (HR) clamped at lactate thresholds 1 and 2 (LT1 and LT2, respectively) were investigated. On separate days, 12 trained males cycled for 15 min at a clamped HR corresponding to LT1 and LT2 under normoxic or hypoxic conditions (simulated altitude of ∼3500 m and inspired oxygen fraction of 13.6%). Power output (PO), arterial oxygen saturation, ventilatory and perceptual responses were measured every 3 min, with metabolic response assessed pre- and post-exercise. At LT1, PO was consistently lower in hypoxia compared to normoxia (p < 0.01). At LT2, PO was not different between normoxia and hypoxia at 3 and 6 min (both p > 0.42) but was significantly lower in hypoxia at 9, 12 and 15 min (all p < 0.04). Overall, hypoxia induced a greater decrease in PO at LT1 (-33.3% ± 11.3%) than at LT2 (-18.0 ± 14.7%) compared to normoxia. Ventilatory, perceptual and metabolic responses were influenced by exercise intensity (all p < 0.01) but not environmental conditions (all p > 0.17). A simulated altitude of ∼3500 m is more effective in reducing cycling PO at LT1 than LT2 during HR clamped cycling while maintaining other internal loads. Therefore, normobaric hypoxia provides a greater benefit via a larger decrease in the mechanical constraints of exercise at lower exercise intensities.

Systemic hypoxia has a larger effect on reducing the external load at lower exercise intensity during .

by Siu Nam Li, Peter Peeling, Brendan R Scott, Jeremiah J Peiffer, Alex Shaykevich and Olivier Girard

Published 2024

European journal of sport science, Early View

The effects of acute hypoxic exposure on mechanical output and internal responses during cycling with heart rate (HR) clamped at lactate thresholds 1 and 2 (LT1 and LT2, respectively) were investigated. On separate days, 12 trained males cycled for 15 min at a clamped HR corresponding to LT1 and LT2 under normoxic or hypoxic conditions (simulated altitude of ∼3500 m and inspired oxygen fraction of 13.6%). Power output (PO), arterial oxygen saturation, ventilatory and perceptual responses were measured every 3 min, with metabolic response assessed pre- and post-exercise. At LT1, PO was consistently lower in hypoxia compared to normoxia (p < 0.01). At LT2, PO was not different between normoxia and hypoxia at 3 and 6 min (both p > 0.42) but was significantly lower in hypoxia at 9, 12 and 15 min (all p < 0.04). Overall, hypoxia induced a greater decrease in PO at LT1 (-33.3% ± 11.3%) than at LT2 (-18.0 ± 14.7%) compared to normoxia. Ventilatory, perceptual and metabolic responses were influenced by exercise intensity (all p < 0.01) but not environmental conditions (all p > 0.17). A simulated altitude of ∼3500 m is more effective in reducing cycling PO at LT1 than LT2 during HR clamped cycling while maintaining other internal loads. Therefore, normobaric hypoxia provides a greater benefit via a larger decrease in the mechanical constraints of exercise at lower exercise intensities.

An analysis of the demands associated with wheelchair basketball

General

Grant no. 19948.

Brendan Scott, Jeremiah Peiffer

An analysis of the demands associated with wheelchair basketball

General

Grant no. 19948.

Brendan Scott, Jeremiah Peiffer

An analysis of the demands associated with wheelchair basketball

General

Grant no. 19948.

Brendan Scott, Jeremiah Peiffer

An analysis of the demands associated with wheelchair basketball

General

Grant no. 19948.

Brendan Scott, Jeremiah Peiffer

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